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Epithelial-Mesenchymal Plasticity (EMP) Impact on Cancer Cell Properties

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Description: Cancer cells resistant to TGF-β’s tumor-inhibitory effects can activate EMT in response to TGF-β signaling, hypoxia or chemotherapy (A). EMT upregulation grants epithelial–mesenchymal plasticity (EMP), allowing cells to exhibit mixed epithelial and mesenchymal traits via EMT pathway modulation, including autocrine TGF-β regulation and TIME interaction (B). EMP enhances cancer cell survival and proliferation under stress (C). Adapted from Figure 1, Proactive and reactive roles of TGF-β in cancer.

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What does the Epithelial-Mesenchymal Plasticity (EMP) Impact on Cancer Cell Properties template show?

Cancer cells resistant to TGF-β’s tumor-inhibitory effects can activate EMT in response to TGF-β signaling, hypoxia or chemotherapy (A). EMT upregulation grants epithelial–mesenchymal plasticity (EMP), allowing cells to exhibit mixed epithelial and mesenchymal traits via EMT pathway modulation, including autocrine TGF-β regulation and TIME interaction (B). EMP enhances cancer cell survival and proliferation under stress (C). Adapted from Figure 1, Proactive and reactive roles of TGF-β in cancer.

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