BioRender-Vorlage

FBXL17 Loss of Function Leads to Increased Global O-GlcNAcylation in Breast Cancer

Erstellt von

Bethany Mason

•

BioRender Team

Diese Vorlage verwenden – Kostenlos

Für die Registrierung ist keine Kreditkarte erforderlich.

Diese Vorlage ist in BioRender vollständig bearbeitbar

Tauschen Sie Text, Formen und Icons einfach aus
Jedes Icon, jede Linie und jedes Label lässt sich umfärben und neu anordnen
Exportieren oder teilen für jeden Anwendungszweck

Schließen Sie sich über 4 Millionen Wissenschaftlern an, die bereits publikationsreife Abbildungen erstellen

Description: The F-box protein encoded by FBXL17 is rearranged in breast cancer. Resulting loss of function in Fbxl17. Uap1, an enzyme involved in O-GlcNAcylation, could interact with Fbxl17. Fbxl17 binds to UAP1 directly and inhibits its phosphorylation, which regulates UAP1 activity. Knockdown of Fbxl17 expression elevated O-GlcNAcylation in breast cancer cells, arguing for a functional role for Fbxl17 in this metabolic pathway.

BioRender-Icons und -Vorlagen entsprechen den Standards für Fachzeitschriften, Förderanträge und Urheberrechte. Bei KI-generierten Abbildungen variieren die Richtlinien. Mehr erfahren

Nicht die passende Vorlage gefunden? Entdecken Sie mehr.

Durchsuchen Sie Tausende von Vorlagen aus über 30 Bereichen der Biowissenschaften.

Beliebte Themen:

Cell

Krebs

Wirkstofffreisetzung

Wirkmechanismus

Signalweg

Versuchen Sie es mit einem Synonym oder registrieren Sie sich, um in der App eine Vorlage vorzuschlagen.

Neueste Vorlagen

AAV Production by Triple Transfection

His-tagged Protein Purification

MHC-Associated Peptide Proteomics (MAPPs)

ChIP sequencing

How Soap Kills COVID-19

Direct ELISA

Whole Genome Sequencing

Restriction Enzymes Cloning

BioRender-Vorlagen-FAQs

What does the FBXL17 Loss of Function Leads to Increased Global O-GlcNAcylation in Breast Cancer template show?

The F-box protein encoded by FBXL17 is rearranged in breast cancer. Resulting loss of function in Fbxl17. Uap1, an enzyme involved in O-GlcNAcylation, could interact with Fbxl17. Fbxl17 binds to UAP1 directly and inhibits its phosphorylation, which regulates UAP1 activity. Knockdown of Fbxl17 expression elevated O-GlcNAcylation in breast cancer cells, arguing for a functional role for Fbxl17 in this metabolic pathway.

Is the FBXL17 Loss of Function Leads to Increased Global O-GlcNAcylation in Breast Cancer template free to use?

Yes, you can customize this template with a free BioRender account (no credit card required). Paid plans unlock additional export formats, higher-resolution downloads, and publication permissions.

Can I edit or customize this template?

Yes, every element is fully editable. Icons, labels, colors, arrows, shapes and layout can all be changed to match your preferences or needs using BioRender's drag-and-drop editor. You can add or remove elements to fully customize your use case.

Is this template accepted for use in journals, theses, or grant applications?

Yes, BioRender icons and templates meet journal, grant, and copyright standards for publication, and many researchers cite BioRender directly in their figure legends.

How do I cite BioRender if I use this template?

Unlock publication permissions with a paid plan, and include 'Created with BioRender.com' in your figure legend, or cite BioRender directly per your journal's guidelines.

What is FBXL17 Loss of Function Leads to Increased Global O-GlcNAcylation in Breast Cancer used for / who typically uses this template?

Researchers and scientists use this template for publications, funding proposals, grant applications, presentations, posters, and more.