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Epithelial-Mesenchymal Transition (EMT) Upregulation and Outcomes
Creator
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Description
EMT facilitates detachment of epithelial cells during wound healing or cancer invasion by altering adhesion, polarity, and promoting mesenchymal traits through factors like SNAI, ZEB1, and TWIST. EMT drives metastasis, enabling cancer cells to invade, intravasate, and become CSCs via FOXC2 signaling, gaining plasticity for metastatic progression. Adapted from Figure 2 of the article 'Spatial and Temporal Relationship between Epithelial–Mesenchymal Transition (EMT) and Stem Cells in Cancer'.
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