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Expression of SELP gene in sickle cell disease patients during crisis

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Parul Gupta

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Description: In sickle cell disease, hemolysis triggers the production of inflammatory molecules, including IL-4 and IL that activates the Jak/STAT signaling pathway and SELP gene expression. Additionally, Bcl-3 plays a critical role in this process. It binds to p52 homodimers at the κB site, resulting in the activation of gene expression. On the other hand, when Bcl-3 binds to p50 homodimers at the κB site, it causes them to separate from DNA, allowing p52 homodimers to take their place.

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In sickle cell disease, hemolysis triggers the production of inflammatory molecules, including IL-4 and IL that activates the Jak/STAT signaling pathway and SELP gene expression. Additionally, Bcl-3 plays a critical role in this process. It binds to p52 homodimers at the κB site, resulting in the activation of gene expression. On the other hand, when Bcl-3 binds to p50 homodimers at the κB site, it causes them to separate from DNA, allowing p52 homodimers to take their place.

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