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This template shows how H. pylori infection of the gastric mucosa can lead to increased PD-L1 and CD58 expression on cancer cells with mutant KRAS, which engage PD1 and CD2 receptors on helper T cells to inhibit Th17 responses. It shows that reduced Th17-derived cytokines suppress anti-tumor immune cells such as cytotoxic T cells, NK cells, neutrophils, and dendritic cells, and that H. pylori elimination can restore this cytokine signaling.
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This template shows how H. pylori infection of the gastric mucosa can lead to increased PD-L1 and CD58 expression on cancer cells with mutant KRAS, which engage PD1 and CD2 receptors on helper T cells to inhibit Th17 responses. It shows that reduced Th17-derived cytokines suppress anti-tumor immune cells such as cytotoxic T cells, NK cells, neutrophils, and dendritic cells, and that H. pylori elimination can restore this cytokine signaling.