BioRender Template

KRAS G12C inhibitor monotherapy

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Kenji Morimoto

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Description: This template shows AXL receptor signaling triggered by Gas6 binding, activating AKT and ERK on one branch and RAS-GTP driving YAP/TAZ nuclear translocation on the other, with a KRAS-G12C inhibitor blocking the RAS-GDP to RAS-GTP conversion. Nuclear YAP/TAZ partners with TEAD to drive transcription that produces more Gas6, which is secreted back out to re-engage AXL at the cell membrane.

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What does the KRAS G12C inhibitor monotherapy template show?

This template shows AXL receptor signaling triggered by Gas6 binding, activating AKT and ERK on one branch and RAS-GTP driving YAP/TAZ nuclear translocation on the other, with a KRAS-G12C inhibitor blocking the RAS-GDP to RAS-GTP conversion. Nuclear YAP/TAZ partners with TEAD to drive transcription that produces more Gas6, which is secreted back out to re-engage AXL at the cell membrane.

Is the KRAS G12C inhibitor monotherapy template free to use?

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