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Neuronal alteration in Huntington's disease associated with GABAergic transmission

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Rebeca Amorim pires

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Description: Mutations in the HTT gene cause abnormal expansion (>37 repeats) of CAG trinucleotide, leading to mutant huntingtin protein accumulation in cells, toxic to neurons. Glutamate (NMDA) receptor dysfunction and GABAA receptor alterations contribute to neuronal hyperexcitability. Inflammatory molecules exacerbate brain inflammation, worsening neuronal damage.

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Mutations in the HTT gene cause abnormal expansion (>37 repeats) of CAG trinucleotide, leading to mutant huntingtin protein accumulation in cells, toxic to neurons. Glutamate (NMDA) receptor dysfunction and GABAA receptor alterations contribute to neuronal hyperexcitability. Inflammatory molecules exacerbate brain inflammation, worsening neuronal damage.

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