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BioRender Template
Role of Endothelial Dysfunction in Heart Failure
Description
This template shows how reduced eNOS activity and nitric oxide in the endothelium lead to reactive oxygen species overproduction, decreased ATR1/ATR2 signaling, and NF-kB driven increases in ICAM1 and VCAM1. These changes result in downstream effects on cardiomyocytes and endothelial permeability, including myocardial damage, fibrosis, hypertrophy, and reduced exercise tolerance.
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