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Summary of the impact of Dars2 deletion on dietary lipid processing in enterocytes
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Description
Deletion of Dars2 in enterocytes leads to increased accumulation of LDs and inhibited lipid transport to peripheral organs. Dars2 deletion in IECs significantly reduces the levels of respiratory chain subunits, and enhances PLIN2 expression, thereby promoting LD formation within enterocytes. Moreover, dietary lipids accumulate and CM release is decreased because of the reduced levels of ApoB48 and impaired Golgi architecture in Dars2IEC-KO mice.
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