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The specific pathogenic mechanism of LRG involvement in neuroinflammation.
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Description
LRG signaling pathways and its regulation expression. TLR4 and several TLR4-mediated inflammatory factors and the LPS released by Gramnegative
bacteria can drive the expression of LRG by activating different transcription factors. Moreover, the combined stimulation of different
cytokines has a synergistic effect on LRG promoter activity. LRG can promote the expression of inflammatory cells through TGF-β signaling and can
also directly promote CSF accumulation through the NF-κB-SPAK pathway.
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